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The 'Treg paradox' in inflammatory arthritis.

  • Julia T Schnell
  • , Raquel Laza Briviesca
  • , Taehyeung Kim
  • , Louis-Marie Charbonnier
  • , Lauren A Henderson
  • , Femke van Wijk
  • , Peter A Nigrovic*
  • *Corresponding author for this work

Research output: Contribution to journalReview articlepeer-review

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Abstract

Classic regulatory T (T reg) cells expressing CD4 and the hallmark transcription factor FOXP3 are integral to the prevention of multi-system autoimmunity. However, immune-mediated arthritis is often associated with increased numbers of T reg cells in the inflamed joints. To understand these seemingly conflicting observations, which we collectively describe as 'the T reg paradox', we provide an overview of T reg cell biology with a focus on T reg cell heterogeneity, function and dysfunction in arthritis. We discuss how the inflamed environment constrains the immunosuppressive activity of T reg cells while also promoting the differentiation of T H17-like T reg cell, exT reg cell (effector T cells that were formerly T reg cells), and osteoclastogenic T reg cell subsets that mediate tissue injury. We present a new framework to understand T reg cells in joint inflammation and define potential strategies for T reg cell-directed interventions in human inflammatory arthritis.

Original languageEnglish
Pages (from-to)9-21
Number of pages13
JournalNature Reviews. Rheumatology
Volume21
Issue number1
Early online date9 Dec 2024
DOIs
Publication statusPublished - Jan 2025

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