The role of the disulfide bond in the interaction of islet amyloid polypeptide with membranes

L.P. Khemtemourian, M.F.M. Engel, J.A.W. Kruijtzer, J.W.M. Hoppener, R.M.J. Liskamp, J.A. Killian

    Research output: Contribution to journalArticleAcademicpeer-review

    Abstract

    Human islet amyloid polypeptide (hIAPP) forms amyloid fibrils in pancreatic islets of patients with type 2 diabetes mellitus. It has been suggested that the N-terminal part, which contains a conserved intramolecular disulfide bond between residues 2 and 7, interacts with membranes, ultimately leading to membrane damage and b-cell death. Here, we used variants of the hIAPP1–19 fragment and model membranes of phosphatidylcholine and phosphatidylserine (7:3, molar ratio) to examine the role of this disulfide in membrane interactions. We found that the disulfide bond has a minor effect on membrane insertion properties and peptide conformational behavior, as studied by monolayer techniques, 2H NMR, ThT-fluorescence, membrane leakage, and CD spectroscopy. The results suggest that the disulfide bond does not play a significant role in hIAPP–membrane interactions. Hence, the fact that this bond is conserved is most likely related exclusively to the biological activity of IAPP as a hormone
    Original languageEnglish
    Pages (from-to)1359-1364
    Number of pages6
    JournalEuropean Biophysics Journal
    Volume39
    Publication statusPublished - 2010

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