RANTES- and interleukin-8-induced responses in normal human eosinophils: Effects of priming with interleukin-5

René C. Schweizer, Berris A C Welmers, Jan A M Raaijmakers, Pieter Zanen, Jan Willem J Lammers, Leo Koenderman*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

140 Citations (Scopus)

Abstract

We report that responses of normal human eosinophils toward the chemokines RANTES and interleukin-8 (IL-8) are modulated and upregulated by priming with IL-5. In a modified Boyden chamber assay, we studied migratory responses toward the members of the chemokine family RANTES, monocyte chemotactic protein-1 (MCP-1), and macrophage inflammatory protein-1α (MIP-1α) (C-C subfamily), and IL-8, platelet factor-4 (PF-4), and neutrophil-activating peptide-2 (NAP-2) (C-x-C subfamily). These chemokines were also studied in terms of actin polymerization and ([Ca2+](i))-mobilizing properties, intracellular signals that are thought to play a role during migratory responses. We found that eosinophils showed significant migratory responses toward RANTES and IL-8 at concentrations of 10-9 to 10-7 mol/L only after priming with IL-5 (10 pmol/L). At these concentrations, PF-4, NAP-2, MCP-1, and MIP-1α induced no significant migratory responses after priming. Unprimed eosinophils only showed a significant migratory response toward RANTES (10-6 mol/L). Changes in [Ca2+](i) were found after addition of RANTES, MIP-1α, and NAP-2 (10 nmol/L) to unprimed eosinophils. RANTES (10- 9 to 10-7 mol/L) significantly induced actin polymerization both in primed and unprimed eosinophils, whereas IL-8 (10-9 to 10-8 mol/L) and MIP-1α (10-8 mol/L) only induced actin polymerization after priming with IL-5. NAP-2, PF-4, and MCP-1 did not affect actin polymerization. These findings are further evidence for the hypothesis that cytokines like IL-5 and locally secreted chemokines like RANTES and IL-8 are both at the basis of specific eosinophil influx into the allergic inflammatory locus.

Original languageEnglish
Pages (from-to)3697-3704
Number of pages8
JournalBlood
Volume83
Issue number12
Publication statusPublished - 15 Jun 1994

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