miR-17-3p contributes to exercise-induced cardiac growth and protects against myocardial ischemia-reperfusion injury

Jing Shi, Yihua Bei, Xiangqing Kong*, Xiaojun Liu, Zhiyong Lei, Tianzhao Xu, Hui Wang, Qinkao Xuan, Ping Chen, Jiahong Xu, Lin Che, Hui Liu, Jiuchang Zhong, Joost P.G. Sluijter, Xinli Li, Anthony Rosenzweig, Junjie Xiao

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

10 Citations (Scopus)
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Abstract

Limited microRNAs (miRNAs, miRs) have been reported to be necessary for exercise-induced cardiac growth and essential for protection against pathological cardiac remodeling. Here we determined members of the miR-17-92 cluster and their passenger miRNAs expressions in two distinct murine exercise models and found that miR-17-3p was increased in both. miR-17-3p promoted cardiomyocyte hypertrophy, proliferation, and survival. TIMP-3 was identified as a direct target gene of miR-17-3p whereas PTEN was indirectly inhibited by miR-17-3p. Inhibition of miR-17-3p in vivo attenuated exercise-induced cardiac growth including cardiomyocyte hypertrophy and expression of markers of myocyte proliferation. Importantly, mice injected with miR-17-3p agomir were protected from adverse remodeling after cardiac ischemia/reperfusion injury. Collectively, these data suggest that miR-17-3p contributes to exercise-induced cardiac growth and protects against adverse ventricular remodeling. miR-17-3p may represent a novel therapeutic target to promote functional recovery after cardiac ischemia/reperfusion.

Original languageEnglish
Pages (from-to)664-676
Number of pages13
JournalTheranostics
Volume7
Issue number3
DOIs
Publication statusPublished - 2017

Keywords

  • Cardiac growth
  • Exercise
  • microRNA

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