KRS: A cut away from release in exosomes

Catherine Rabouille*

*Corresponding author for this work

Research output: Contribution to journalComment/Letter to the editorAcademicpeer-review

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Abstract

Cancer cells often trigger an inflammatory process, which in some cases may be driven by the presence of lysyltRNA synthetase (KRS) in the medium. Kim et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201605118) now demonstrate that cleavage of the KRS by caspase-8 inside cells triggers its interaction with syntenin and its release in inflammatory exosomes.

Original languageEnglish
Pages (from-to)1891-1893
Number of pages3
JournalJournal of Cell Biology
Volume216
Issue number7
DOIs
Publication statusPublished - 1 Jul 2017

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