Abstract
Burns proposes an intriguing hypothesis by suggesting that the "schizophrenia genes" might not be regulatory genes themselves, but rather closely associated with regulatory genes directly involved in the proper growth of the social brain. We point out that this account would benefit from incorporating the effects of localized lesions and aberrant hemispheric asymmetry on cortical connectivity underlying the social brain. In addition, we argue that the evolutionary framework is superfluous.
Original language | English |
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Number of pages | 1 |
Journal | Behavioral and Brain Sciences |
Volume | 27 |
Issue number | 6 |
DOIs | |
Publication status | Published - 1 Dec 2004 |