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Fcγ receptor IIIA genotype is associated with rituximab response in antimyelin-associated glycoprotein neuropathy

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

BACKGROUND: Treatment with anti-B cell antibody rituximab may ameliorate the disease course in a subgroup of patients with polyneuropathy associated with IgM monoclonal gammopathy. Polymorphisms of leukocyte IgG receptors (FcγR) that influence efficiency of antibody-dependent cell-mediated cytotoxicity determine rituximab efficacy in patients with lymphoma and autoimmune disease.

OBJECTIVE: To investigate the association of FcγRIIA and FcγRIIIA polymorphisms with the response to rituximab treatment in a cohort of patients with polyneuropathy associated with IgM monoclonal gammopathy (PNP-IgM) with and without antimyelin-associated glycoprotein antibodies.

METHODS: We determined FcγRIIA-R/H131 and FcγRIIIA-V/F158 genotypes in 27 patients with PNP-IgM using allele-specific PCR and Sanger sequencing.

RESULTS: The FcγRIIIA-V/V158 genotype was associated with functional improvement (p=0.02) after 1 year.

CONCLUSIONS: FcγRIIIA polymorphisms are potential biomarkers for response to rituximab treatment in polyneuropathy associated with IgM monoclonal gammopathy.

Original languageEnglish
Pages (from-to)918-920
Number of pages3
JournalJournal of Neurology, Neurosurgery and Psychiatry
Volume85
Issue number8
DOIs
Publication statusPublished - Aug 2014

Keywords

  • Adult
  • Aged
  • Antibodies, Monoclonal, Murine-Derived
  • Cohort Studies
  • Demyelinating Diseases
  • Female
  • Glycoproteins
  • Humans
  • Male
  • Middle Aged
  • Myelin Sheath
  • Netherlands
  • Neural Conduction
  • Paraproteinemias
  • Polymorphism, Genetic
  • Polyneuropathies
  • Prospective Studies
  • Receptors, IgG
  • Rituximab
  • Treatment Outcome
  • Journal Article
  • Research Support, Non-U.S. Gov't

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