CRISPR-Based Adenine Editors Correct Nonsense Mutations in a Cystic Fibrosis Organoid Biobank

Maarten H. Geurts, Eyleen de Poel, Gimano D. Amatngalim, Rurika Oka, Fleur M. Meijers, Evelien Kruisselbrink, Peter van Mourik, Gitte Berkers, Karin M. de Winter-de Groot, Sabine Michel, Danya Muilwijk, Bente L. Aalbers, Jasper Mullenders, Sylvia F. Boj, Sylvia W.F. Suen, Jesse E. Brunsveld, Hettie M. Janssens, Marcus A. Mall, Simon Y. Graeber, Ruben van BoxtelCornelis K. van der Ent, Jeffrey M. Beekman*, Hans Clevers

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

2 Citations (Scopus)

Abstract

Adenine base editing (ABE) enables enzymatic conversion from A-T into G-C base pairs. ABE holds promise for clinical application, as it does not depend on the introduction of double-strand breaks, contrary to conventional CRISPR/Cas9-mediated genome engineering. Here, we describe a cystic fibrosis (CF) intestinal organoid biobank, representing 664 patients, of which ~20% can theoretically be repaired by ABE. We apply SpCas9-ABE (PAM recognition sequence: NGG) and xCas9-ABE (PAM recognition sequence: NGN) on four selected CF organoid samples. Genetic and functional repair was obtained in all four cases, while whole-genome sequencing (WGS) of corrected lines of two patients did not detect off-target mutations. These observations exemplify the value of large, patient-derived organoid biobanks representing hereditary disease and indicate that ABE may be safely applied in human cells.

Original languageEnglish
Pages (from-to)503-510.e7
JournalCell stem cell
Volume26
Issue number4
DOIs
Publication statusPublished - 2 Apr 2020

Keywords

  • adenine base-editing
  • Cas9 off-target analysis
  • CFTR mutations
  • CRISPR/Cas9
  • cystic fibrosis
  • evolved Cas9 proteins
  • genome editing
  • human intestinal organoids
  • organoid biobank
  • patient-derived adult stem cells

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