Accelerated telomere shortening in rheumatic diseases: cause or consequence?

A.Z. Dehbi, T.R.D.J. Radstake, J.C.A. dr. Broen

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

Accelerated aging of the immune system (immune aging), represented by telomere shortening, has been implicated in a variety of rheumatic diseases. Studies addressing telomere shortening in rheumatic diseases so far yielded controversial results. The current review aims to provide an overview on the role of immune aging in a plethora of immune-mediated conditions including systemic sclerosis, rheumatoid arthritis, systemic lupus erythematosus and osteoarthritis. The main question this review aims to answer is whether rheumatic diseases cause accelerated aging or that accelerated aging drives rheumatic diseases.

Original languageEnglish
Pages (from-to)1193-1204
Number of pages12
JournalExpert Review of Clinical Immunology
Volume9
Issue number12
DOIs
Publication statusPublished - Dec 2013

Keywords

  • Aging, Premature
  • Animals
  • Humans
  • Immune System
  • Rheumatic Diseases
  • T-Lymphocytes
  • Telomere Shortening
  • Journal Article
  • Review

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